About: The relationship between human coronaviruses, asthma and allergy

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About: The relationship between human coronaviruses, asthma and allergy – an unresolved dilemma Goto Sponge NotDistinct Permalink

An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	The relationship between human coronaviruses, asthma and allergy – an unresolved dilemma
Creator	Kowalski, Marek Chałubiński, Maciej Gajewski, Adrian
Source	Medline; PMC
abstract	Human coronaviruses (HCoVs) such as HCoV‐229E or OC43 are responsible for mild upper airway infections, whereas highly pathogenic HCoVs, including SARS‐CoV, MERS‐CoV and SARS‐CoV‐2, often evoke acute, heavy pneumonias. They tend to induce immune responses based on interferon and host inflammatory cytokine production and promotion of T1 immune profile. Less is known about their effect on T2‐type immunity. Unlike human rhinoviruses (HRV) and rhinosyncytioviruses (RSV), HCoVs are not considered as a dominant risk factor of severe exacerbations of asthma, mostly T2‐type chronic inflammatory disease. The relationship between coronaviruses and T2‐type immunity, especially in asthma and allergy, is not well understood. This review aims to summarise currently available knowledge about the relationship of HCoVs, including novel SARS‐CoV‐2, with asthma and allergic inflammation.
has issue date	2020-08-06(xsd:dateTime)
bibo:doi	10.1111/cea.13718
bibo:pmid	32762099
has license	no-cc
sha1sum (hex)	0518338a4ffd3384f14e259030b95bd07f71900c
schema:url	https://doi.org/10.1111/cea.13718
resource representing a document's title	The relationship between human coronaviruses, asthma and allergy – an unresolved dilemma
has PubMed Central identifier	PMC7436768
has PubMed identifier	32762099
schema:publication	Clin Exp Allergy
resource representing a document's body	covid:0518338a4ffd3384f14e259030b95bd07f71900c#body_text
is schema:about of	named entity 'immune' named entity 'highly' named entity 'mild' named entity 'DPP4' named entity 'melanoma' named entity 'HOST' named entity 'RHINOVIRUSES' named entity 'PATHOGENIC' named entity 'MILD' named entity 'CURRENTLY' named entity 'CHRONIC INFLAMMATORY DISEASE' named entity 'KNOWN' named entity 'HIGHLY' named entity 'CORONAVIRUSES' named entity 'AIMS' named entity 'IMMUNE RESPONSES' named entity 'HEAVY' named entity 'KNOWLEDGE' named entity 'RSV' named entity 'MERS-CoV' named entity 'coronaviruses' named entity 'inflammatory disease' named entity 'IFN' named entity 'protein' named entity 'HCoV-229E' named entity 'allergy' named entity 'HRV' named entity 'activated B cells' named entity 'enhancer' named entity 'human coronavirus' named entity 'DPP4' named entity 'light-chain' named entity 'interferon' named entity 'cytokine' named entity 'CD13' named entity 'T-cell' named entity 'asthma' named entity '229E' named entity 'MERS-CoV' named entity 'mucosal' named entity 'lymphopenia' named entity 'pathogenicity' named entity 'cytokine' named entity 'CD26' named entity 'eosinophils' named entity 'RSV' named entity 'ACE-2' named entity 'SARS-CoV' named entity 'allergic inflammation' named entity 'asthma' named entity 'eosinophil' named entity 'DCs' named entity 'IFN-γ' named entity 'COVID19' named entity 'ACE-2' named entity 'immunological' named entity 'G-CSF' named entity 'IFN-γ' named entity 'TMPRSS2' named entity 'lower respiratory tract' named entity 'CD26' named entity 'SARS-CoV' named entity 'asthma' named entity 'cytokine storm' named entity 'SARS-CoV-2' named entity 'NL63' named entity 'RNA viruses' named entity 'OC43' named entity 'intensive care unit' named entity 'serum' named entity 'asthma exacerbations' named entity 'HRV' named entity 'HCoV-OC43' named entity 'cellular receptors' named entity 'sputum' named entity 'monocyte' named entity 'allergy' named entity 'spike proteins' named entity 'plasma'

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