About: Tertiary lymphoid organs in infection and autoimmunity

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
title	Tertiary lymphoid organs in infection and autoimmunity
Creator	Hammad, Hamida Neyt, Katrijn Dé Ric Perros, Fré Geurtsvankessel, Corine Lambrecht, Bart
source	Elsevier; Medline; PMC
abstract	The lymph nodes (LNs) and spleen have an optimal structure that allows the interaction between T cells, B cells and antigen-presenting dendritic cells (DCs) on a matrix made up by stromal cells. Such a highly organized structure can also be formed in tertiary lymphoid organs (TLOs) at sites of infection or chronic immune stimulation. This review focuses on the molecular mechanisms of TLO formation and maintenance, the controversies surrounding the nature of the inducing events, and the functions of these structures in infection, transplantation and autoimmunity.
has issue date	2012-05-21(xsd:dateTime)
bibo:doi	10.1016/j.it.2012.04.006
bibo:pmid	22622061
has license	no-cc
sha1sum (hex)	1659ac3a5ce90d49d5c3e21ad79dbd41458ba404
schema:url	https://doi.org/10.1016/j.it.2012.04.006
resource representing a document's title	Tertiary lymphoid organs in infection and autoimmunity
has PubMed Central identifier	PMC7106385
has PubMed identifier	22622061
schema:publication	Trends Immunol
resource representing a document's body	covid:1659ac3a5ce90d49d5c3e21ad79dbd41458ba404#body_text
is schema:about of	named entity 'LYMPHOID ORGANS' named entity 'INFECTION' named entity 'TERTIARY' named entity 'AUTOIMMUNITY' named entity 'chemokines' named entity 'pDCs' named entity 'innate lymphoid cells' named entity 'Trends in Immunology' named entity 'lymphatics' named entity 'Th17 cells' named entity 'lung' named entity 'FDCs' named entity 'adenovirus' named entity 'myofibroblast' named entity 'immunoglobulin' named entity 'systemic lupus erythematosus' named entity 'mice' named entity 'chemokine' named entity 'transplant rejection' named entity 'extravasation' named entity 'adaptive immune responses' named entity 'morbidity' named entity 'lymphoid organs' named entity 'afferent' named entity 'cell type' named entity 'milky spots' named entity 'LTo' named entity 'atherosclerosis' named entity 'mice' named entity 'spleen' named entity 'evolution' named entity 'SLE' named entity 'orphan receptor' named entity 'innate lymphoid cells' named entity 'commensals' named entity 'ectopic' named entity 'FRCs' named entity 'homeostatic' named entity 'Id2' named entity 'TLOs' named entity 'evolutionary pressure' named entity 'stromal cells' named entity 'IL-7' named entity 'lymphoid tissue' named entity 'mice' named entity 'lymphatics' named entity 'autoimmune' named entity 'lymphocytes' named entity 'lymphotoxin' named entity 'peritoneal cavity' named entity 'thyroid' named entity 'postnatal development' named entity 'cigarette smoke' named entity 'infection' named entity 'effector cells' named entity 'CXCR5' named entity 'CD4 T cell' named entity 'autoantigen' named entity 'autoimmune reactions' named entity 'diabetes' named entity 'TNF' named entity 'immune response' named entity 'tumor necrosis factor' named entity 'DCs' named entity 'LTbR' named entity 'antigen-presenting cells' named entity 'NOD1' named entity 'SLOs' named entity 'SLOs' named entity 'influenza viruses' named entity 'influenza virus' named entity 'virus' named entity 'inflammation' named entity 'CCL20' named entity 'lung' named entity 'SARS' named entity 'antigen-presenting'

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