About: Angiotensin receptor blockers and COVID-19

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Angiotensin receptor blockers and COVID-19
Source	Elsevier; Medline; PMC; WHO
abstract	Abstract Angiotensin Receptor Blockers (ARBs) exhibit major pleiotropic protecting effects beyond their antihypertensive properties, including reduction of inflammation. ARBs directly protect the lung from the severe acute respiratory syndrome as a result of viral infections, including those from coronavirus. The protective effect of ACE2 is enhanced by ARB administration. For these reasons ARB therapy must be continued for patients affected by hypertension, diabetes and renal disease, comorbidities of the current COVID-19 pandemic. Controlled clinical studies should be conducted to determine whether ARBs may be included as additional therapy for COVID-19 patients.
has issue date	2020-06-30(xsd:dateTime)
bibo:doi	10.1016/j.phrs.2020.104832
bibo:pmid	32304747
has license	els-covid
sha1sum (hex)	ac7267dfe0d8e4bb44c72a6f0e4d19dd28b4845c
schema:url	https://doi.org/10.1016/j.phrs.2020.104832
resource representing a document's title	Angiotensin receptor blockers and COVID-19
has PubMed Central identifier	PMC7158830
has PubMed identifier	32304747
schema:publication	Pharmacological Research
resource representing a document's body	covid:ac7267dfe0d8e4bb44c72a6f0e4d19dd28b4845c#body_text
is schema:about of	named entity 'infection' named entity 'antihypertensive' named entity 'receptor' named entity 'ACE2' named entity 'AT1R' named entity 'epithelial' named entity 'SARS-CoV-2' named entity 'mitochondrial function' named entity 'ACE2' named entity 'ARBs' named entity 'Ang1' named entity 'negative regulator' named entity 'cardiovascular disorders' named entity 'Pharmacological' named entity 'ARBs' named entity 'Angiotensin' named entity 'ARBs' named entity 'reduce inflammation' named entity 'COVID-19' named entity 'endothelial' named entity 'COVID' named entity 'ACE2' named entity 'COVID' named entity 'complement activation' named entity 'high mobility Group' named entity 'cross-protective' named entity 'ARBs' named entity 'clinical evidence' named entity 'COVID' named entity 'hypertension' named entity 'coagulation' named entity 'ACE2' named entity 'mesenchymal stem cells' named entity 'scientific evidence' named entity 'fibrosis' named entity 'ARBs' named entity 'infection' named entity 'enzymatic activity' named entity 'COVID' named entity 'MSCs' named entity 'NFκB' named entity 'severe acute respiratory syndrome' named entity 'COVID' named entity 'ACE2' named entity 'COVID' named entity 'kinase' named entity 'upregulation' named entity 'virus' named entity 'Transforming Growth Factor Beta' named entity 'ROS' named entity 'ARBs' named entity 'SARS-CoV-2' named entity 'coronavirus' named entity 'Angiotensin Receptor Blockers' named entity 'apoptosis' named entity 'COVID-19 pandemic' named entity 'infection' named entity 'receptor' named entity 'ACE2' named entity 'metabolic disorders' named entity 'lung' named entity 'antihypertensives' named entity 'ARBs' named entity 'proinflammatory' named entity 'lung disease' named entity 'congestive heart failure' named entity 'Angiotensin II' named entity 'inflammation' named entity 'SARS-CoV-2' named entity 'comorbidities' named entity 'ARBs' named entity 'endothelial dysfunction' named entity 'lung' named entity 'renal function' named entity 'ARBs' named entity 'Controlled clinical studies' named entity 'SARS-CoV-2' named entity 'Ebola outbreak in Africa' named entity 'ARBs' named entity 'HMGB1' named entity 'hypertension' named entity 'viral infections'

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