About: Alveolar epithelium and Na,K-ATPase in acute lung injury

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Alveolar epithelium and Na,K-ATPase in acute lung injury
Creator	Sznajder, Jacob Raviv, Stacy Sznajder, J Vadász, István Raviv, · Vadász, I
Source	PMC
abstract	Active transport of sodium across the alveolar epithelium, undertaken in part by the Na,K-adenosine triphosphatase (Na,K-ATPase), is critical for clearance of pulmonary edema fluid and thus the outcome of patients with acute lung injury. Acute lung injury results in disruption of the alveolar epithelial barrier and leads to impaired clearance of edema fluid and altered Na,K-ATPase function. There has been significant progress in the understanding of mechanisms regulating alveolar edema clearance and signaling pathways modulating Na,K-ATPase function during lung injury. The accompanying review by Morty et al. focuses on intact organ and animal models as well as clinical studies assessing alveolar fluid reabsorption in alveolar epithelial injury. Elucidation of the mechanisms underlying regulation of active Na(+) transport, as well as the pathways by which the Na,K-ATPase regulates epithelial barrier function and edema clearance, are of significance to identify interventional targets to improve outcomes of patients with acute lung injury.
has issue date	2007-05-25(xsd:dateTime)
bibo:doi	10.1007/s00134-007-0661-8
bibo:pmid	17530222
has license	no-cc
sha1sum (hex)	b2662d9c7cb6deb8dd5b47e3e2155b6ce7d0aca3
schema:url	https://doi.org/10.1007/s00134-007-0661-8
resource representing a document's title	Alveolar epithelium and Na,K-ATPase in acute lung injury
has PubMed Central identifier	PMC7095466
has PubMed identifier	17530222
schema:publication	Intensive Care Med
resource representing a document's body	covid:b2662d9c7cb6deb8dd5b47e3e2155b6ce7d0aca3#body_text
is schema:about of	named entity 'epithelial' named entity 'fluid' named entity 'fluid' named entity 'models' named entity 'mechanisms' named entity 'lung injury' named entity 'barrier function' named entity 'regulates' named entity 'epithelial' named entity 'sodium' named entity 'improve' named entity 'disruption' named entity 'acute lung injury' named entity 'reabsorption' named entity 'acute lung injury' named entity 'Na,K-ATPase' named entity 'Na,K-ATPase' named entity 'clinical studies' named entity 'organ' named entity 'edema' named entity 'lung injury' named entity 'Alveolar epithelium' named entity 'BASIC RESEARCH' named entity 'dopamine' named entity 'ALI' named entity 'sodium' named entity 'alveolar spaces' named entity 'high-energy phosphate' named entity 'protein' named entity 'gas exchange' named entity 'β-adrenergic' named entity 'exocytosis' named entity 'wound healing' named entity 'gas exchange' named entity 'Morbidity and mortality' named entity 'KGF' named entity 'alveolar epithelial cells' named entity 'protein phosphatase 2A' named entity 'intercellular adhesion' named entity '100% oxygen' named entity 'Na,K-ATPase' named entity 'U0126' named entity 'gradient' named entity 'alveolar epithelium' named entity 'actin cytoskeleton' named entity 'structurally similar' named entity 'Na,K-ATPase' named entity 'polypeptide' named entity 'Raf-1' named entity 'alveolar epithelium' named entity 'hypoxemic' named entity 'inactive form' named entity 'corticosteroids' named entity 'mRNA' named entity 'plasma membrane' named entity 'alveolar epithelium' named entity 'adhesion molecules' named entity 'intracellular' named entity 'oxidative stress' named entity 'Coagulation' named entity 'epithelial' named entity 'Na,K-ATPase' named entity 'tidal volume' named entity 'cDNA' named entity 'PKC-δ' named entity 'interstitium' named entity 'Na,K-ATPase' named entity 'ERK' named entity 'solutes' named entity 'lung injury' named entity 'reperfusion injury' named entity 'Na,K-ATPase' named entity 'catecholamines' named entity 'tissue factor pathway inhibitor' named entity 'glycosylated' named entity 'hypocapnic'

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