About: Antiviral T-cell therapy

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Antiviral T-cell therapy
Creator	Brenner, Malcolm Heslop, Helen Leen, Ann
Source	Medline; PMC
abstract	Serious viral infections are a common cause of morbidity and mortality after allogeneic stem cell transplantation. They occur in the majority of allograft recipients and are fatal in 17–20%. These severe infections may be prolonged or recurrent and add substantially to the cost, both human and financial, of the procedure. Many features of allogeneic stem cell transplantation contribute to this high rate of viral disease. The cytotoxic and immunosuppressive drugs administered pre-transplant to eliminate the host hematopoietic/immune system and any associated malignancy, the delay in recapitulating immune ontogeny post-transplant, the immunosuppressive drugs given to prevent graft versus host disease (GvHD), and the effects of GvHD itself, all serve to make stem cell transplant recipients vulnerable to disease from endogenous (latent) and exogenous (community) viruses, and to be incapable of controlling them as quickly and effectively as most normal individuals.
has issue date	2014-02-11(xsd:dateTime)
bibo:doi	10.1111/imr.12138
bibo:pmid	24517423
has license	green-oa
sha1sum (hex)	d948a23a78546aab029c795211438116ece8f57f
schema:url	https://doi.org/10.1111/imr.12138
resource representing a document's title	Antiviral T-cell therapy
has PubMed Central identifier	PMC3927231
has PubMed identifier	24517423
schema:publication	Immunological Reviews
resource representing a document's body	covid:d948a23a78546aab029c795211438116ece8f57f#body_text
is schema:about of	named entity 'T-cell' named entity 'Antiviral' named entity 'virus' named entity 'HLA' named entity 'skin rash' named entity 'EBV' named entity 'CD4 +' named entity 'great strides' named entity 'protein' named entity 'HLA' named entity 'EBV' named entity 'GvHD' named entity 'immunodominant' named entity 'allograft' named entity 'cytokines' named entity 'Alloantigen' named entity 'peptide' named entity 'TCR' named entity 'epitope' named entity 'monoclonal antibodies' named entity 'antiviral activity' named entity 'viral vectors' named entity 'VSTs' named entity 'VSTs' named entity 'HSCT' named entity 'VST' named entity '12 weeks' named entity 'VSTs' named entity 'virus' named entity 'immunosuppression' named entity 'CMV' named entity 'CMV' named entity 'adenoviral vector' named entity 'class I HLA' named entity 'viruses' named entity 'rash' named entity 'antigens' named entity 'viral antigens' named entity 'endogenous' named entity 'Guinan' named entity 'prophylaxis' named entity 'cytokine' named entity 'CD4 +' named entity 'antigens' named entity 'gene product' named entity 'GvHD' named entity 'VST' named entity 'homeostatic mechanisms' named entity 'HLA' named entity 'dimerize' named entity 'VSTs' named entity 'HLA' named entity 'EBV' named entity 'endogenous' named entity 'antiviral agents' named entity 'effector T cells' named entity 'epitopes' named entity 'HLA' named entity 'antiviral' named entity 'transplant recipients' named entity 'CMV' named entity 'EBNA1' named entity 'viral infections' named entity 'CD4 +' named entity 'immunogenic' named entity 'CMV' named entity 'amino acids' named entity 'cytotoxic' named entity 'CD8 +' named entity 'effector' named entity 'haplotypes' named entity 'APC' named entity 'antigens' named entity 'virus' named entity 'EBV' named entity 'CMV' named entity 'gastroenteritis' named entity 'EBV' named entity 'HLA'

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