About: The requirement of environmental acidification for Ibaraki virus infection to host cells

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About: The requirement of environmental acidification for Ibaraki virus infection to host cells Goto Sponge NotDistinct Permalink

An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	The requirement of environmental acidification for Ibaraki virus infection to host cells
Creator	Iwata, Hiroyuki Watanabe, Rie Shibutani, Shusaku Tsuruta, Yuya
Source	Medline; PMC
abstract	The effect of environmental acidification on Ibaraki virus (IBAV) infection was tested using endosomal inhibitory chemicals and low pH treatment. Treatment of target cells with endosomal inhibitors significantly decreased the progeny virus production. IBAV outer capsid proteins, VP5 and VP2, were removed from virion when purified IBAV was exposed to low pH environment. Further experiment showed that the exposure to low pH buffer facilitated IBAV infection when the cellular endosomal pathway was impaired by bafilomycin A1. Results obtained in this study suggest that acidic environment is essential to initiate IBAV infection.
has issue date	2015-08-28(xsd:dateTime)
bibo:doi	10.1292/jvms.15-0222
bibo:pmid	26321298
has license	cc-by-nc-nd
sha1sum (hex)	5d352d8eaadb17dc1ed4cd7501e3533d366c939c
schema:url	https://doi.org/10.1292/jvms.15-0222
resource representing a document's title	The requirement of environmental acidification for Ibaraki virus infection to host cells
has PubMed Central identifier	PMC4751137
has PubMed identifier	26321298
schema:publication	J Vet Med Sci
resource representing a document's body	covid:5d352d8eaadb17dc1ed4cd7501e3533d366c939c#body_text
is schema:about of	named entity 'TREATMENT' named entity 'SUGGEST' named entity 'DECREASED' named entity 'USING' named entity 'ACIDIFICATION' named entity 'OBTAINED' named entity 'CELLULAR' named entity 'LOW PH' named entity 'ENVIRONMENT' named entity 'EXPOSURE TO' named entity 'ENVIRONMENTAL' named entity 'PURIFIED' named entity 'CHEMICALS' named entity 'STUDY' named entity 'VP5' named entity 'INHIBITORS' named entity 'VIRION' named entity 'INFECTION' named entity 'ENDOSOMAL PATHWAY' named entity 'HOST CELLS' named entity 'ACIDIFICATION' named entity 'VIRUS INFECTION' named entity 'REQUIREMENT' named entity 'BAFILOMYCIN A1' named entity 'ENVIRONMENTAL' named entity 'INITIATE' named entity 'ESSENTIAL' named entity 'TARGET CELLS' named entity 'PROGENY' named entity 'EXPOSED TO' named entity 'VP2' named entity 'PH BUFFER' named entity 'IMPAIRED' named entity 'INHIBITORY' named entity 'EXPERIMENT' named entity 'IBARAKI VIRUS' named entity 'IBARAKI VIRUS' named entity 'OUTER CAPSID' named entity 'EFFECT' named entity 'ACIDIC ENVIRONMENT' named entity 'RESULTS' named entity 'REMOVED' named entity 'VIRUS PRODUCTION' named entity 'TESTED' named entity 'CAPSID PROTEINS' covid:arg/5d352d8eaadb17dc1ed4cd7501e3533d366c939c named entity 'purified' named entity 'virus' named entity 'Treatment' named entity 'initiate' named entity 'host' named entity 'bafilomycin A1' named entity 'VP5' named entity 'pH buffer' named entity 'acidification' named entity 'Results' named entity 'experiment' named entity 'clathrin-dependent endocytosis' named entity 'low pH' named entity 'significant effect' named entity 'influenza virus' named entity 'endosomal pathway' named entity 'low pH' named entity 'infection' named entity 'Ibaraki disease' named entity 'serotype' named entity 'infection' named entity 'adaptor protein' named entity 'PBS' named entity 'infectivity' named entity 'PBS' named entity 'endosome' named entity 'infection' named entity 'endosomal pathway' named entity 'chlorpromazine' named entity 'genus' named entity 'causative agent' named entity 'orbivirus'

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