About: Relevance of signaling molecules for apoptosis induction on influenza A virus replication

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Relevance of signaling molecules for apoptosis induction on influenza A virus replication
Creator	Miyazaki, Tadaaki Iwai, Atsushi Shiozaki, Takuya
Source	Elsevier; Medline; PMC
abstract	Abstract Apoptosis is an important mechanism to maintain homeostasis in mammals, and disruption of the apoptosis regulation mechanism triggers a range of diseases, such as cancer, autoimmune diseases, and developmental disorders. The severity of influenza A virus (IAV) infection is also closely related to dysfunction of apoptosis regulation. In the virus infected cells, the functions of various host cellular molecules involved in regulation of induction of apoptosis are modulated by IAV proteins to enable effective virus replication. The modulation of the intracellular signaling pathway inducing apoptosis by the IAV infection also affects extracellular mechanisms controlling apoptosis, and triggers abnormal host responses related to the disease severity of IAV infections. This review focuses on apoptosis related molecules involved in IAV replication and pathogenicity, the strategy of the virus propagation through the regulation of apoptosis is also discussed.
has issue date	2013-11-22(xsd:dateTime)
bibo:doi	10.1016/j.bbrc.2013.10.100
bibo:pmid	24177013
has license	els-covid
sha1sum (hex)	6694f16b82f96d357255e5bc174f8e70137c7de9
schema:url	https://doi.org/10.1016/j.bbrc.2013.10.100
resource representing a document's title	Relevance of signaling molecules for apoptosis induction on influenza A virus replication
has PubMed Central identifier	PMC7092955
has PubMed identifier	24177013
schema:publication	Biochemical and Biophysical Research Communications
resource representing a document's body	covid:6694f16b82f96d357255e5bc174f8e70137c7de9#body_text
is schema:about of	named entity 'strategy' named entity 'regulation' named entity 'regulation' named entity 'host' named entity 'ABNORMAL' named entity 'SEVERITY' named entity 'EXTRACELLULAR' named entity 'INFECTION' named entity 'INFECTED' named entity 'HOMEOSTASIS' named entity 'FUNCTIONS' named entity 'CELLS' named entity 'EFFECTIVE' named entity 'REPLICATION' named entity 'DISEASE SEVERITY' named entity 'PATHOGENICITY' named entity 'INDUCTION OF APOPTOSIS' named entity 'VARIOUS' named entity 'MAMMALS' named entity 'HOST' named entity 'DEVELOPMENTAL DISORDERS' named entity 'CANCER' named entity 'DISEASES' named entity 'TRIGGERS' named entity 'IMPORTANT' named entity 'APOPTOSIS' named entity 'DISRUPTION' named entity 'REGULATION' named entity 'CELLULAR' named entity 'MODULATED' named entity 'TO MAINTAIN' named entity 'RESPONSES' named entity 'DYSFUNCTION' named entity 'MECHANISM' named entity 'REGULATION OF APOPTOSIS' named entity 'SIGNALING MOLECULES' named entity 'VIRUS REPLICATION' named entity 'INTRACELLULAR SIGNALING PATHWAY' named entity 'INVOLVED' named entity 'REVIEW' named entity 'RELEVANCE' named entity 'INFLUENZA A VIRUS' named entity 'APOPTOSIS INDUCTION' named entity 'INFLUENZA A VIRUS' named entity 'MOLECULES' named entity 'RELATED' named entity 'RANGE' named entity 'MODULATION' named entity 'VIRUS REPLICATION' named entity 'STRATEGY' named entity 'INDUCING' named entity 'CONTROLLING' named entity 'INFECTIONS' named entity 'PROTEINS' named entity 'AUTOIMMUNE DISEASES' named entity 'MECHANISMS' named entity 'ENABLE' named entity 'VIRUS' named entity 'PROPAGATION' named entity 'focuses' named entity 'This' named entity 'modulated' named entity 'dysfunction' named entity 'mechanism' named entity 'infection' named entity 'mammals' named entity 'diseases' named entity 'IAV' named entity 'Apoptosis' named entity 'intracellular signaling' named entity 'infection' named entity 'IAV' named entity 'apoptosis' named entity 'influenza A virus' named entity 'apoptosis' named entity 'virus' named entity 'homeostasis' named entity 'apoptosis' named entity 'homeostasis'

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