About: Immunological Aspects of Systemic Vasculitis

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Immunological Aspects of Systemic Vasculitis
Creator	Ahmed, Melika Louzir, Hechmi
Source	PMC
abstract	Primary vasculitis are commonly multifactorial disorders involving environmental, genetic and immunological factors. Several immune-based effector mechanisms are implicated in the vascular wall damage. These effector mechanisms commonly imply auto-antibodies or immune complexes - mediated cytotoxicity but the contribution of a T-cell mediated immune response has also been described, particularly in large vascular vasculitis. Despite advances in understanding the pathophysiological mechanisms of vasculitis, the triggering events initiating the disease remain largely undefined in most cases. This review highlights the recent advances in the etiopathogenesis of primary vasculitis. A better understanding of the immunological aspects of these disorders may provide insight into the development of novel therapeutical strategies.
has issue date	2011-10-12(xsd:dateTime)
bibo:doi	10.1007/174_2011_144
has license	no-cc
sha1sum (hex)	6accec2bc9cea5716a88c218a3976f4fd8c9a90c
schema:url	https://doi.org/10.1007/174_2011_144
resource representing a document's title	Immunological Aspects of Systemic Vasculitis
has PubMed Central identifier	PMC7121773
schema:publication	Systemic Vasculitis
resource representing a document's body	covid:6accec2bc9cea5716a88c218a3976f4fd8c9a90c#body_text
is schema:about of	named entity 'SYSTEMIC VASCULITIS' named entity 'IMMUNOLOGICAL' named entity 'SLE' named entity 'macrophages' named entity 'cytotoxic T lymphocytes' named entity 'immune complexes' named entity 'elastic arteries' named entity 'Polymorphism' named entity 'perforin' named entity 'antigens' named entity 'endothelial cells' named entity 'MPO' named entity 'c-ANCA' named entity 'infection' named entity 'ANCA' named entity 'genetic factors' named entity 'Parvovirus B19' named entity 'ITPKC' named entity 'Cytotoxicity' named entity 'pro-inflammatory cytokines' named entity 'GCA' named entity 'antibody' named entity 'aorta' named entity 'antigenic' named entity 'extravascular' named entity 'kidneys' named entity 'upregulation' named entity 'cytotoxicity' named entity 'vasculitis' named entity 'endothelial cells' named entity 'vascular lesions' named entity 'HBV' named entity 'parvovirus B19' named entity 'vascular disease' named entity 'MPO' named entity 'antibody' named entity 'ANCA' named entity 'lungs' named entity 'GCA' named entity 'complement pathway' named entity 'biopsies' named entity 'vasculitis' named entity 'ANCA' named entity 'co-trimoxazole' named entity 'IFN' named entity 'asthma' named entity 'GCA' named entity 'basophil' named entity 'HSP' named entity 'endothelial cells' named entity 'vasculitis' named entity 'co-stimulatory' named entity 'DCs' named entity 'mesangium' named entity 'vascular wall' named entity 'CSS' named entity 'TLR4' named entity 'neutrophils' named entity 'genetic predisposition' named entity 'immunology' named entity 'macrophages' named entity 'serum' named entity 'Fujieda' named entity 'epidemiological studies' named entity 'autoimmunity' named entity 'heterogeneous disorders' named entity 'polymorphism' named entity 'CSS' named entity 'CD4 +' named entity 'autoantibodies' named entity 'anti-CD20' named entity 'autoantibody' named entity 'IL-17' named entity 'IgA' named entity 'Neutrophils' named entity 'GCA' named entity 'macrophages' named entity 'HUV' named entity 'immune complexes' named entity 'DCs' named entity 'glomerulonephritis' named entity 'HBV'

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