About: Endoplasmic reticulum stress-induced apoptosis in the development of reproduction

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About: Endoplasmic reticulum stress-induced apoptosis in the development of reproduction Goto Sponge NotDistinct Permalink

An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Endoplasmic reticulum stress-induced apoptosis in the development of reproduction
Creator	Cheng', Weng-Jun Dar', Chun-Fan Inr, Kang-Sheng Pen<p, Zheng-Hang Tong', Hua
topic	covid:80cd55670315eb1716da214e6a546cb3c4c34d24#this
Source	Elsevier; Medline; PMC
abstract	Proteins synthesized in the endoplasmic reticulum (ER) are properly folded with the assistance of ER chaperones. Accumulation of misfolded protein in the ER triggers an adaptive ER stress (ERS) response termed the unfolded protein response. Recent interest has focused on the possibility that the accumulation of misfolded proteins can also contribute to reproductive response, including preimplantation embryos, testicular germ cell, placenta, and unexplained intrauterine growth restriction (IUGR). The major ERS pathway constituents are present at all stages of preimplantation development and that the activation of ERS pathways can be induced at the 8-cell, morula and blastocyst stage. This review mainly introduced the research progress of ERS induced apoptosis of reproductive cells, providing a new direction for the research of reproductive disease therapy.
has issue date	2016-03-31(xsd:dateTime)
bibo:doi	10.7669/j.issn.1001-7844.2016.01.0051
bibo:pmid	32288402
has license	els-covid
sha1sum (hex)	80cd55670315eb1716da214e6a546cb3c4c34d24
schema:url	https://doi.org/10.7669/j.issn.1001-7844.2016.01.0051
resource representing a document's title	Endoplasmic reticulum stress-induced apoptosis in the development of reproduction
has PubMed Central identifier	PMC7129369
has PubMed identifier	32288402
schema:publication	Journal of Reproduction and Contraception
resource representing a document's body	covid:80cd55670315eb1716da214e6a546cb3c4c34d24#body_text
is http://vocab.deri.ie/void#inDataset of	proxy:http/ns.inria.fr/covid19/80cd55670315eb1716da214e6a546cb3c4c34d24
is schema:about of	named entity 'ERS' named entity 'Proteins' named entity 'chaperones' named entity 'direction' named entity 'development' named entity 'reproductive cells' named entity 'DEVELOPMENT' named entity 'REPRODUCTION' named entity 'ACCUMULATION' named entity 'REVIEW' named entity 'FOCUSED' named entity 'ERS' named entity 'POSSIBILITY' named entity 'ER STRESS' named entity 'CONTRIBUTE ' named entity 'GERM CELL' named entity 'PATHWAY' named entity 'INCLUDING' named entity 'CONSTITUENTS' named entity 'UNEXPLAINED' named entity '8-CELL' named entity 'PREIMPLANTATION EMBRYOS' named entity 'THERAPY' named entity 'DEVELOPMENT' named entity 'PROTEINS' named entity 'RECENT' named entity 'RESPONSE' named entity '80%' named entity 'STAGE' named entity 'APOPTOSIS' named entity 'BLASTOCYST' named entity 'REVIEW' named entity 'REPRODUCTIVE DISEASE' named entity 'RESEARCH' named entity 'UNFOLDED PROTEIN RESPONSE' named entity 'PLACENTA' named entity 'ENDOPLASMIC RETICULUM STRESS-INDUCED APOPTOSIS' named entity 'ACTIVATION' named entity 'SYNTHESIZED' named entity 'REPRODUCTIVE' named entity 'FOLDED' named entity 'PRESENT' named entity 'ALL STAGES' named entity 'INDUCED' named entity 'INTRODUCED' named entity 'TESTICULAR' named entity 'INTRAUTERINE GROWTH RESTRICTION' named entity 'ASSISTANCE' named entity 'ENDOPLASMIC RETICULUM' named entity 'TRIGGERS' named entity 'PATHWAYS' named entity 'PROGRESS' named entity 'MORULA' named entity 'INTEREST' named entity 'NEW' named entity 'DIRECTION' named entity 'PROVIDING' named entity 'MAJOR' named entity 'ADAPTIVE' named entity 'induced' named entity 'possibility' named entity 'proteins' named entity 'The major' named entity 'disease' named entity 'testicular' named entity 'apoptosis' named entity 'testicular' named entity 'reproductive cells' named entity 'intrauterine growth restriction' named entity 'cadmium' named entity 'signaling pathways' named entity 'placental development' named entity 'kinase' named entity 'preeclampsia' named entity 'apoptosis'

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