About: Redox control in the pathophysiology of influenza virus infection

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
title	Redox control in the pathophysiology of influenza virus infection
Creator	Lin, Chang-Shen Yokoyama, Kazunari Chen, Ker-Kong Ku, Chia-Chen Kuo, Kung-Kai Lin, Ying-Chu Minakuchi, Moeko Pan, Jia-Bin Saito, Shigeo Wuputra, Kenly
source	PMC
abstract	Triggered in response to external and internal ligands in cells and animals, redox homeostasis is transmitted via signal molecules involved in defense redox mechanisms through networks of cell proliferation, differentiation, intracellular detoxification, bacterial infection, and immune reactions. Cellular oxidation is not necessarily harmful per se, but its effects depend on the balance between the peroxidation and antioxidation cascades, which can vary according to the stimulus and serve to maintain oxygen homeostasis. The reactive oxygen species (ROS) that are generated during influenza virus (IV) infection have critical effects on both the virus and host cells. In this review, we outline the link between viral infection and redox control using IV infection as an example. We discuss the current state of knowledge on the molecular relationship between cellular oxidation mediated by ROS accumulation and the diversity of IV infection. We also summarize the potential anti-IV agents available currently that act by targeting redox biology/pathophysiology.
has issue date	2020-07-20(xsd:dateTime)
bibo:doi	10.1186/s12866-020-01890-9
has license	cc-by
sha1sum (hex)	8c15c122463361963f01da670c29650a1781f884
schema:url	https://doi.org/10.1186/s12866-020-01890-9
resource representing a document's title	Redox control in the pathophysiology of influenza virus infection
has PubMed Central identifier	PMC7370268
schema:publication	BMC Microbiol
resource representing a document's body	covid:8c15c122463361963f01da670c29650a1781f884#body_text
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