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About:
[Case Report] Stress induced (Takotsubo) cardiomyopathy triggered by the COVID-19 pandemic
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schema:ScholarlyArticle
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wasabi.inria.fr
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Type:
Academic Article
research paper
schema:ScholarlyArticle
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type
Academic Article
research paper
schema:ScholarlyArticle
isDefinedBy
Covid-on-the-Web dataset
has title
[Case Report] Stress induced (Takotsubo) cardiomyopathy triggered by the COVID-19 pandemic
Creator
Spandidos, Demetrios
Manolis, Athanasios
Rizos, Emmanouil
Triantafyllis, Andreas
Tsamakis, Konstantinos
Tsiptsios, Dimitrios
Giannitsi, Sofia
Kallistratos, Manolis
Poulimenos, Leonidas
Tsinivizov, Pavlos
Varvarousis, Dimitris
Source
Medline; PMC
abstract
Stress induced (Takotsubo) cardiomyopathy (TC) represents an acute heart failure syndrome triggered by physical or emotional stressors. COVID-19 pandemic has caused an unprecedented health crisis resulting in fear, distress and anxiety, with emerging cardiovascular implications. COVID-19 related stress can act as potential trigger for TC. We present a case of an elderly female who developed TC due to stress surrounding COVID-19.
has issue date
2020-07-07
(
xsd:dateTime
)
bibo:doi
10.3892/etm.2020.8968
bibo:pmid
32765776
has license
cc-by-nc-nd
sha1sum (hex)
933dc2b43781acd12523d7ecabc1e67924e7ea87
schema:url
https://doi.org/10.3892/etm.2020.8968
resource representing a document's title
[Case Report] Stress induced (Takotsubo) cardiomyopathy triggered by the COVID-19 pandemic
has PubMed Central identifier
PMC7401702
has PubMed identifier
32765776
schema:publication
Exp Ther Med
resource representing a document's body
covid:933dc2b43781acd12523d7ecabc1e67924e7ea87#body_text
is
schema:about
of
named entity 'pandemic'
named entity 'COVID-19'
named entity 'acute heart failure'
named entity 'blood pressure'
named entity 'IL-6'
named entity 'Electrocardiogram'
named entity 'systolic'
named entity 'stressor'
named entity 'adrenal gland'
named entity 'diastole'
named entity 'catecholamine'
named entity 'COVID-19 pandemic'
named entity 'HPA'
named entity 'left ventricular'
named entity 'myocardial stunning'
named entity 'biomarker'
named entity 'systemic inflammatory response'
named entity 'left ventricular'
named entity 'physical stress'
named entity 'sinus rhythm'
named entity 'oxygen saturation'
named entity 'cardiomyopathy'
named entity 'left ventricular'
named entity 'morbidity'
named entity 'inflammatory markers'
named entity 'adrenal medulla'
named entity 'ventriculography'
named entity 'aspirin'
named entity 'systole'
named entity 'CXCL1'
named entity 'hypokinesia'
named entity 'pathognomonic'
named entity 'interleukin-6'
named entity 'ventriculography'
named entity 'statin'
named entity 'neuroendocrine'
named entity 'stenotic'
named entity 'TTE'
named entity 'heart rate'
named entity 'stressor'
named entity 'blood tests'
named entity 'β2-adrenergic receptor'
named entity 'significant factor'
named entity 'troponin'
named entity 'pathogenesis'
named entity 'cortisol'
named entity 'macrophage'
named entity 'electrocardiographic findings'
named entity 'ejection fraction'
named entity 'angiography'
named entity 'anxiety'
named entity 'Clinical presentation'
named entity 'COVID-19'
named entity 'ACS'
named entity 'adrenal cortex'
named entity 'medical history'
named entity 'anxiety'
named entity 'ACS'
named entity 'IL-8'
named entity 'Left ventricular'
named entity 'ST segment elevation'
named entity 'acute coronary syndrome'
named entity 'sympathetic activity'
named entity 'Overstimulation'
named entity 'acute heart failure'
named entity 'cardiomyopathy'
named entity 'COVID-19'
named entity 'pro-inflammatory'
named entity 'end-systolic'
named entity 'serum'
named entity 'CD16'
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