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About:
ACE1 polymorphism and progression of SARS
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An Entity of Type :
schema:ScholarlyArticle
, within Data Space :
wasabi.inria.fr
associated with source
document(s)
Type:
Academic Article
research paper
schema:ScholarlyArticle
New Facet based on Instances of this Class
Attributes
Values
type
Academic Article
research paper
schema:ScholarlyArticle
isDefinedBy
Covid-on-the-Web dataset
has title
ACE1 polymorphism and progression of SARS
Creator
Hijikata, Minako
Keicho, Naoto
Matsushita, Ikumi
Kawana, Akihiko
Itoyama, Satoru
Kirikae, Teruo
Kuratsuji, Tadatoshi
Ohashi, Jun
Quy, Tran
Sasazuki, Takehiko
Yanai, Hideki
Dang Ha, Le
Long, Hoang
Phi, Nguyen
Van Ban, Vo
Source
Elsevier; Medline; PMC
abstract
Abstract We have hypothesized that genetic predisposition influences the progression of SARS. Angiotensin converting enzyme (ACE1) insertion/deletion (I/D) polymorphism was previously reported to show association with the adult respiratory distress syndrome, which is also thought to play a key role in damaging the lung tissues in SARS cases. This time, the polymorphism was genotyped in 44 Vietnamese SARS cases, with 103 healthy controls who had had a contact with the SARS patients and 50 controls without any contact history. SARS cases were divided into either non-hypoxemic or hypoxemic groups. Despite the small sample size, the frequency of the D allele was significantly higher in the hypoxemic group than in the non-hypoxemic group (p =0.013), whereas there was no significant difference between the SARS cases and controls, irrespective of a contact history. ACE1 might be one of the candidate genes that influence the progression of pneumonia in SARS.
has issue date
2004-10-22
(
xsd:dateTime
)
bibo:doi
10.1016/j.bbrc.2004.08.208
bibo:pmid
15381116
has license
els-covid
sha1sum (hex)
a26310448a2c11a83217bbe892b56c20534a7223
schema:url
https://doi.org/10.1016/j.bbrc.2004.08.208
resource representing a document's title
ACE1 polymorphism and progression of SARS
has PubMed Central identifier
PMC7092806
has PubMed identifier
15381116
schema:publication
Biochemical and Biophysical Research Communications
resource representing a document's body
covid:a26310448a2c11a83217bbe892b56c20534a7223#body_text
is
schema:about
of
named entity 'POLYMORPHISM'
named entity 'ACE1'
named entity 'adult respiratory distress syndrome'
named entity 'sample size'
named entity 'Despite'
named entity 'patients'
named entity 'frequency'
named entity 'controls'
named entity 'TIME'
named entity 'EITHER'
named entity 'SAMPLE SIZE'
named entity 'KEY'
named entity 'ANGIOTENSIN CONVERTING ENZYME'
named entity 'CASES'
named entity 'SARS'
named entity 'ALLELE'
named entity 'PATIENTS'
covid:arg/a26310448a2c11a83217bbe892b56c20534a7223
named entity 'THOUGHT'
named entity 'REPORTED'
named entity 'POLYMORPHISM'
named entity 'PREVIOUSLY'
named entity 'LUNG TISSUES'
named entity 'VIETNAMESE'
named entity 'SIGNIFICANT'
named entity 'FREQUENCY'
named entity 'CONTACT'
named entity '28P'
named entity 'ACE1'
named entity 'ADULT RESPIRATORY DISTRESS SYNDROME'
named entity 'PROGRESSION'
named entity '28I'
named entity 'DIVIDED'
named entity 'HEALTHY'
named entity 'CANDIDATE GENES'
named entity 'PLAY'
named entity 'DAMAGING'
named entity '103'
named entity 'SMALL'
named entity 'INFLUENCE'
named entity 'MIGHT BE'
named entity 'CONTACT WITH'
named entity 'ASSOCIATION'
named entity 'HISTORY'
named entity 'GENETIC PREDISPOSITION'
named entity 'PNEUMONIA'
named entity 'SARS'
named entity 'PROGRESSION'
named entity 'ROLE'
named entity 'HAVE'
named entity 'HIGHER'
named entity 'ONE OF'
named entity 'GROUP'
named entity 'INSERTION'
named entity 'GROUPS'
named entity 'NON-'
named entity 'DIFFERENCE'
named entity 'influences'
named entity 'genetic predisposition'
named entity 'hypoxemic'
named entity 'influence'
named entity 'pneumonia'
named entity 'sample size'
named entity 'Angiotensin converting enzyme'
named entity 'hypoxemic'
named entity 'polymorphism'
named entity 'allele'
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