About: MHV-A59 coronavirus infection produces a transient episode of demyelination that is followed by spontaneous remyelination. This paradigm provides a complex lesion environment to examine cellular and molecular mechanisms involved in successful CNS remyelination. Our work in this model has focused on the roles of platelet-derived growth factor and fibroblast growth factor 2 in regulating oligodendrocyte progenitor responses required for remyelination.   Goto Sponge  NotDistinct  Permalink

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  • MHV-A59 coronavirus infection produces a transient episode of demyelination that is followed by spontaneous remyelination. This paradigm provides a complex lesion environment to examine cellular and molecular mechanisms involved in successful CNS remyelination. Our work in this model has focused on the roles of platelet-derived growth factor and fibroblast growth factor 2 in regulating oligodendrocyte progenitor responses required for remyelination.
subject
  • Neuroscience
  • Metabolism
  • Neurology
  • Central nervous system
  • Growth factors
  • Glial cells
  • Anatomical pathology
  • Medical signs
  • Nervous tissue cells
  • Neurohistology
  • Protein domains
  • World Anti-Doping Agency prohibited substances
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