About: Virus et sensibilisation

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An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	Virus et sensibilisation
Creator	Brouard, J Bessière, A Nimal, D
Source	Elsevier; Medline; PMC
abstract	Résumé Les virus respiratoires peuvent, en se répliquant aussi bien au niveau de l’épithélium respiratoire supérieur qu’inférieur, induire des sifflements. La plupart des études cliniques soulignent qu’une symptomatologie sifflante secondaire à l’atteinte par un virus respiratoire durant la prime enfance est liée à un risque ultérieur d’asthme. Les virus peuvent induire une exacerbation asthmatique par un effet direct sur sa cible principale, l’épithélium, mais aussi d’une réaction immuno-inflammatoire médiée par voie systémique. Par ailleurs, des études ont également montré des résultats discordants dans la capacité d’une infection virale d’induire des réponses allergiques significatives. Est-ce le virus l’initiateur de l’asthme allergique ou les sifflements viro-induits le révélateur d’enfants déjà prédisposés à l’atopie en raison d’anomalies préexistantes pulmonaires et/ou de la réponse immunitaire ? Abstract Respiratory viruses are able to replicate in both the upper and lower respiratory epithelium, thus inducing wheezing. Most clinical studies suggest that wheezing illnesses due to respiratory viruses in early childhood are linked to a risk for asthma. Viruses may induce asthma exacerbations through direct effects on their main target, the respiratory epithelium, as well as via a systemic immune-inflammatory reaction. On other hand, studies have also yielded conflicting results in regard to the ability of viral infection to enhance subsequent allergic responses. Do early viral illnesses cause allergic asthma or viral wheezing episodes serve to reveal children who are already predisposed to this disease on basis of an abnormal lung physiology and/or immune response?
has issue date	2009-04-30(xsd:dateTime)
bibo:doi	10.1016/j.reval.2009.01.021
bibo:pmid	32288888
has license	els-covid
sha1sum (hex)	b7a554771811099404c0fc1660ead288841cf081
schema:url	https://doi.org/10.1016/j.reval.2009.01.021
resource representing a document's title	Virus et sensibilisation
has PubMed Central identifier	PMC7102747
has PubMed identifier	32288888
schema:publication	Revue Française d'Allergologie
resource representing a document's body	covid:b7a554771811099404c0fc1660ead288841cf081#body_text
is schema:about of	named entity 'ont' named entity 'INFECTION VIRALE' named entity 'SUR' named entity 'SAS' named entity 'UNE' named entity 'DIRECT' named entity 'VIRUS' named entity 'SECONDAIRE' named entity 'CLINIQUES' named entity 'DES' named entity 'EXACERBATION' named entity 'EFFET' named entity 'MAIS' named entity 'VIRUS' named entity 'RESPIRATOIRE' named entity 'SUP' named entity 'PRIME' named entity 'ONT' named entity 'EST' named entity 'SENSITIZATION' named entity 'PAR' named entity 'LES' named entity 'INFLAMMATOIRE' named entity 'MASSON' named entity 'virus' named entity 'risque' named entity 'rapprocher' named entity 'dont' named entity 'IL-4' named entity 'permis' named entity 'rhinovirus' named entity 'Asthma' named entity 'IgE' named entity 'IgE' named entity 'rhinovirus' named entity 'rhinovirus' named entity 'allergie' named entity 'Elsevier Masson' named entity 'le révélateur' named entity 'par' named entity 'Par' named entity 'mais' named entity 'Elsevier Masson' named entity 'voie' named entity 'virus' named entity 'prime' named entity 'direct' named entity 'par' named entity 'Résumé' named entity 'sur' named entity 'infection' named entity 'virus' named entity 'dans' named entity 'Virus' named entity 'Tous' named entity 'par' named entity 'virus' named entity 'Virus' named entity 'sensitization'
is part of of	Virus et sensibilisation Résumé Les virus respiratoires peuvent, en se répliquant aussi bien au niveau de l’épithélium respiratoire supérieur qu’inférieur, induire des sifflements. La plupart des études cliniques soulignent qu’une symptomatologie sifflante secondaire à l’atteinte par un virus respiratoire durant la prime enfance est liée à un risque ultérieur d’asthme. Les virus peuvent induire une exacerbation asthmatique par un effet direct sur sa cible principale, l’épithélium, mais aussi d’une réaction immuno-inflammatoire médiée par voie systémique. Par ailleurs, des études ont également montré des résultats discordants dans la capacité d’une infection virale d’induire des réponses allergiques significatives. Est-ce le virus l’initiateur de l’asthme allergique ou les sifflements viro-induits le révélateur d’enfants déjà prédisposés à l’atopie en raison d’anomalies préexistantes pulmonaires et/ou de la réponse immunitaire ? Abstract Respiratory viruses are able to replicate in both the upper and lower respiratory epithelium, thus inducing wheezing. Most clinical studies suggest that wheezing illnesses due to respiratory viruses in early childhood are linked to a risk for asthma. Viruses may induce asthma exacerbations through direct effects on their main target, the respiratory epithelium, as well as via a systemic immune-inflammatory reaction. On other hand, studies have also yielded conflicting results in regard to the ability of viral infection to enhance subsequent allergic responses. Do early viral illnesses cause allergic asthma or viral wheezing episodes serve to reveal children who are already predisposed to this disease on basis of an abnormal lung physiology and/or immune response? covid:b7a554771811099404c0fc1660ead288841cf081#body_text

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