About: GSK3 modulation in acute lung injury, myocarditis and polycystic kidney disease-related aneurysm()

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About: GSK3 modulation in acute lung injury, myocarditis and polycystic kidney disease-related aneurysm() Goto Sponge NotDistinct Permalink

An Entity of Type : schema:ScholarlyArticle, within Data Space : wasabi.inria.fr associated with source document(s)

Attributes	Values
type	Academic Article research paper schema:ScholarlyArticle
isDefinedBy	Covid-on-the-Web dataset
has title	GSK3 modulation in acute lung injury, myocarditis and polycystic kidney disease-related aneurysm()
Creator	Chiou, Shih-Hwa Lin, Chiang, Fu-Tien Lin, Heng-Liang Liu, Wei-Lun Chiou, Heng-Liang Jen, Fu Kao, Shih-Hwa Kao, Wah Tze, Juliana Tze-Wah, Juliana
Source	Elsevier; Medline; PMC
abstract	GSK3 are involved in different physical and pathological conditions and inflammatory regulated by macrophages contribute to significant mechanism. Infection stimuli may modulate GSK3 activity and influence host cell adaption, immune cells infiltration or cytokine expressions. To further address the role of GSK3 modulation in macrophages, the signal transduction of three major organs challenged by endotoxin, virus and genetic inherited factors are briefly introduced (lung injury, myocarditis and autosomal dominant polycystic kidney disease). As a result of pro-inflammatory and anti-inflammatory functions of GSK3 in different microenvironments and stages of macrophages (M1/M2), the rational resolution should be considered by adequately GSK3.
has issue date	2020-07-18(xsd:dateTime)
bibo:doi	10.1016/j.bbamcr.2020.118798
bibo:pmid	32693109
has license	no-cc
sha1sum (hex)	ff374d59ff214c16652fe4460a71b4b32ad354bb
schema:url	https://doi.org/10.1016/j.bbamcr.2020.118798
resource representing a document's title	GSK3 modulation in acute lung injury, myocarditis and polycystic kidney disease-related aneurysm()
has PubMed Central identifier	PMC7368652
has PubMed identifier	32693109
schema:publication	Biochim Biophys Acta Mol Cell Res
resource representing a document's body	covid:ff374d59ff214c16652fe4460a71b4b32ad354bb#body_text
is schema:about of	named entity 'die' named entity 'inhibition' named entity 'conditions' named entity 'GSK3β' named entity 'GSK3' named entity 'myocarditis' named entity 'activity' named entity 'mice' named entity 'sensitivity' named entity 'cell' named entity 'inflammatory' named entity 'inherited' named entity 'insulin' named entity 'immune cells' named entity 'lung inflammation' named entity 'interact' named entity 'animal studies' named entity 'protein expression' named entity 'HMGB1' named entity 'M1 macrophages' named entity 'IL-1β' named entity 'phosphorylation' named entity 'Autosomal dominant polycystic kidney disease' named entity 'GSK3' named entity 'podocytes' named entity 'CREB' named entity 'IL-10' named entity 'Macrolides' named entity 'serine/threonine protein kinase' named entity 'regulated' named entity 'PI3K' named entity 'factors' named entity 'GSK3' named entity 'null mice' named entity 'Toll-like receptor' named entity 'pathological conditions' named entity 'PI3K' named entity 'signal transduction' named entity 'cytokine' named entity 'Akt' named entity 'monocytes' named entity 'GSK3' named entity 'insulin sensitivity' named entity 'signaling pathway' named entity 'myocarditis' named entity 'pro-inflammatory' named entity 'polycystic kidney disease' named entity 'aneurysm' named entity 'acute lung injury' named entity 'macrophages' named entity 'GSK3β' named entity 'animal models' named entity 'sepsis' named entity 'transcription' named entity 'knock-in' named entity 'PC1' named entity 'PXN' named entity 'ERK' named entity 'Lithium' named entity 'elastases' named entity 'LRP2' named entity 'macrophages' named entity 'GSK3β' named entity 'endomyocardial biopsy' named entity 'berberine' named entity 'S100A8' named entity 'ADPKD' named entity 'matrix metalloproteinases' named entity 'clinical study' named entity 'alveolar' named entity 'megalin'

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